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Potential effects of Metformin in DNA BER system based on oxidative status in type 2 diabetes
The cells were treated with metformin, and various cellular functions were assessed, including proliferation, apoptosis, migration, and epithelial-mesenchymal transition (EMT) status ( In summary, metformin disrupts the normal proliferation process of glioma cells and triggers apoptosis by activating the AMPK/mTOR signaling pathway, cell cycle arrest, mitochondria-dependent apoptosis pathway, inhibition of tumor cell metabolism, oxidative stress, and DNA damage, which provides a new idea for the treatment of glioma
Vegetable, fruit and meat consumption and potential risk modifying genes in relation to colorectal cancer
Yes, it is generally safe for all skin types
TLRs govern neutrophil activity in aspergillosis
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